Title Alternative | Incisional hernia repair after wide excision of the iliac bone |
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FullText URL | 128_117.pdf |
Author | Tsukuda, Kazunori| Asano, Hiroaki| Mandai, Yasuhiro| Fujiwara, Toshiyoshi| |
Abstract | The patient was a 46-year old Japanese female who had undergone wide excision of the iliac bone and hip transposition at our institute's orthopedics department 2 years earlier. She presented with a growing incisional hernia and was transferred to our gastroenterological surgery department for surgical treatment. We planned a mesh repair for the incisional hernia, which protruded over the right iliac bone. The dimensions of the abdominal defect were 15×9 cm, and we used prolene mesh to repair the defect. The mesh was fixed at the inner part of the iliac bone, folded back at the iliac horn and fixed to the abdominal oblique muscles. The postoperative course was smooth, and recurrence was not seen at 3.5 years after the operation. An incisional hernia as seen in this patient's case is very rare, but we found that the underlay technique and prolene mesh were very useful for the three-dimensional hernia repair. |
Keywords | 腹壁瘢痕ヘルニア(incisional hernia) 腸骨軟骨肉腫(chondrosarcoma of the iliac bone) 腸骨広範囲切除術(wide excision of the iliac bone) プロリーンメッシュ(prolene mesh) |
Publication Title | Journal of Okayama Medical Association |
Published Date | 2016-08-01 |
Volume | volume128 |
Issue | issue2 |
Start Page | 117 |
End Page | 120 |
ISSN | 0030-1558 |
Related Url | isVersionOf https://doi.org/10.4044/joma.128.117 |
language | Japanese |
Copyright Holders | Copyright (c) 2016 岡山医学会 |
File Version | publisher |
DOI | 10.4044/joma.128.117 |
NAID | 130005262528 |
Author | Hayashi, Tatsuro| Asano, Hiroaki| Toyooka, Shinichi| Tsukuda, Kazunori| Soh, Junichi| Shien, Tadahiko| Taira, Naruto| Maki, Yuho| Tanaka, Norimitsu| Doihara, Hiroyoshi| Nasu, Yasutomo| Huh, Nam-ho| Miyoshi, Shinichiro| |
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Published Date | 2012-05 |
Publication Title | Journal of Cancer Research and Clinical Oncology |
Volume | volume138 |
Issue | issue5 |
Content Type | Journal Article |
JaLCDOI | 10.18926/AMO/52140 |
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FullText URL | 68_1_23.pdf |
Author | Ueno, Tsuyoshi| Toyooka, Shinichi| Fukazawa, Takuya| Kubo, Takafumi| Soh, Junichi| Asano, Hiroaki| Muraoka, Takayuki| Tanaka, Norimitsu| Maki, Yuho| Shien, Kazuhiko| Furukawa, Masashi| Sakaguchi, Masakiyo| Yamamoto, Hiromasa| Tsukuda, Kazunori| Miyoshi, Shinichiro| |
Abstract | The microRNA-34s (miR-34s) have p53 response elements in their 5ʼ-flanking regions and demonstrate tumor-suppressive functions. In malignant pleural mesothelioma (MPM), we previously reported that expression of miR-34b and miR-34c (miR-34b/c) was frequently downregulated by methylation in MPM cell lines and primary tumors. The forced overexpression of miR-34b/c showed significant antitumor effects with the induction of apoptosis in MPM cells. In this study, we examined the in vivo antitumor effects of miR-34b/c using adenovirus vector on MPM. We subcutaneously transplanted NCI-H290, a human MPM cell line, into BALB/C mice and injected adenovirus vector expressing miR-34b/c, luciferase driven by the cytomegalovirus promoter (Ad-miR-34b/c or Ad-Luc), or PBS control into tumors over 5mm in diameter. A statistically significant growth inhibition of the tumor volume was observed in the Ad-miR-34b/c group from day 6 onward compared to the Ad-Luc group. The inhibition rate of Ad-miR-34b/c, compared to the tumor volume treated with Ad-Luc, was 58.6% on day 10 and 54.7% on day13. Our results indicate that adenovirus-mediated miR-34b/c gene therapy could be useful for the clinical treatment of MPM. |
Keywords | mesothelioma microRNA microRNA-34b/c p53 |
Amo Type | Original Article |
Publication Title | Acta Medica Okayama |
Published Date | 2014-02 |
Volume | volume68 |
Issue | issue1 |
Publisher | Okayama University Medical School |
Start Page | 23 |
End Page | 26 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
Copyright Holders | CopyrightⒸ 2014 by Okayama University Medical School |
File Version | publisher |
Refereed | True |
PubMed ID | 24553485 |
Web of Science KeyUT | 000331592800004 |
Author | Muraoka, Takayuki| Soh, Junichi| Toyooka, Shinichi| Aoe, Keisuke| Fujimoto, Nobukazu| Hashida, Shinsuke| Maki, Yuho| Tanaka, Norimitsu| Shien, Kazuhiko| Furukawa, Masashi| Yamamoto, Hiromasa| Asano, Hiroaki| Tsukuda, Kazunori| Kishimoto, Takumi| Otsuki, Takemi| Miyoshi, Shinichiro| |
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Published Date | 2013-12 |
Publication Title | Lung Cancer |
Volume | volume82 |
Issue | issue3 |
Content Type | Journal Article |
Author | Maki, Yuho| Soh, Junichi| Ichimura, Kouichi| Shien, Kazuhiko| Furukawa, Masashi| Muraoka, Takayuki| Tanaka, Norimitsu| Ueno, Tsuyoshi| Yamamoto, Hiromasa| Asano, Hiroaki| Tsukuda, Kazunori| Toyooka, Shinichi| Miyoshi, Shinichiro| |
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Published Date | 2013-01 |
Publication Title | Oncology Reports |
Volume | volume29 |
Issue | issue1 |
Content Type | Journal Article |
Author | Ueno, Tsuyoshi| Tsukuda, Kazunori| Toyooka, Shinichi| Ando, Midori| Takaoka, Munenori| Soh, Junichi| Asano, Hiroaki| Maki, Yuho| Muraoka, Takayuki| Tanaka, Norimitsu| Shien, Kazuhiko| Furukawa, Masashi| Yamatsuji, Tomoki| Kiura, Katsuyuki| Naomoto, Yoshio| Miyoshi, Shinichiro| |
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Published Date | 2012-04 |
Publication Title | Lung Cancer |
Volume | volume76 |
Issue | issue1 |
Content Type | Journal Article |
Author | Tanaka, Norimitsu| Toyooka, Shinichi| Soh, Junichi| Kubo, Takafumi| Yamamoto, Hiromasa| Maki, Yuho| Muraoka, Takayuki| Shien, Kazuhiko| Furukawa, Masashi| Ueno, Tsuyoshi| Asano, Hiroaki| Tsukuda, Kazunori| Aoe, Keisuke| Miyoshi, Shinichiro| |
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Published Date | 2012-04 |
Publication Title | Lung Cancer |
Volume | volume76 |
Issue | issue1 |
Content Type | Journal Article |
Author | Shien, Kazuhiko| Toyooka, Shinichi| Ichimura, Kouichi| Soh, Junichi| Furukawa, Masashi| Maki, Yuho| Muraoka, Takayuki| Tanaka, Norimitsu| Ueno, Tsuyoshi| Asano, Hiroaki| Tsukuda, Kazunori| Yamane, Masaomi| Oto, Takahiro| Kiura, Katsuyuki| Miyoshi, Shinichiro| |
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Published Date | 2012-07 |
Publication Title | Lung Cancer |
Volume | volume77 |
Issue | issue1 |
Content Type | Journal Article |
JaLCDOI | 10.18926/AMO/49253 |
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FullText URL | 67_1_19.pdf |
Author | Furukawa, Masashi| Soh, Junichi| Yamamoto, Hiromasa| Ichimura, Kouichi| Shien, Kazuhiko| Maki, Yuho| Muraoka, Takayuki| Tanaka, Norimitsu| Ueno, Tsuyoshi| Asano, Hiroaki| Tsukuda, Kazunori| Toyooka, Shinichi| Miyoshi, Shinichiro| |
Abstract | Nuclear factor of κ-light polypeptide gene enhancer in B cells inhibitor α (NFKBIA), which is a tumor suppressor gene, was found to be silenced in lung adenocarcinomas. We examined NFKBIA expression, mutations in the EGFR and K-ras genes, and EML4-ALK fusion in 101 resected lung adenocarcinoma samples from never-smokers. NFKBIA expression was evaluated using immunohistochemistry. NFKBIA expression was negative in 16 of the 101 samples (15.8%). EGFR and K-ras mutations and EML4-ALK fusion were detected in 61 (60.5%), 1 (1.0%), and 2 (2.0%) of the 101 samples, respectively, in a completely mutually exclusive manner. Negative NFKBIA expression was observed significantly more frequently among the tumors with none of the three genetic alterations compared to those with such alterations (p=0.009). In addition, negative NFKBIA expression was significantly more frequent among the EGFR-wild type samples compared to the EGFR-mutant samples (p=0.013). In conclusion, NFKBIA expression was silenced in adenocarcinomas without EGFR/K-ras mutations or EML4-ALK fusion, suggesting that the silencing of NFKBIA may play an important role in the carcinogenesis of adenocarcinomas independent of EGFR/K-ras mutations or EML4-ALK fusion. |
Keywords | never-smoker lung cancer adenocarcinoma nuclear factor of κ-light polypeptide gene enhancer in B cells inhibitor α epidermal growth factor receptor |
Amo Type | Original Article |
Publication Title | Acta Medica Okayama |
Published Date | 2013-02 |
Volume | volume67 |
Issue | issue1 |
Publisher | Okayama University Medical School |
Start Page | 19 |
End Page | 24 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
Copyright Holders | CopyrightⒸ 2013 by Okayama University Medical School |
File Version | publisher |
Refereed | True |
PubMed ID | 23439505 |
Web of Science KeyUT | 000316829900003 |
Related Url | http://ousar.lib.okayama-u.ac.jp/metadata/52534 |
JaLCDOI | 10.18926/AMO/48967 |
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FullText URL | 66_5_423.pdf |
Author | Furukawa, Masashi| Izumi, Sadanobu| Tsukuda, Kazunori| Tokumo, Masaki| Sakurai, Jun| Mano, Shohey| |
Abstract | An 81-year-old man was found to have a pancreatic head tumor on abdominal computed tomography (CT) performed during a follow-up visit for sigmoid colon cancer. The tumor had a diameter of 35mm on the CT scan and was diagnosed as pancreatic head carcinoma T3N0M0. The patient was treated with pylorus-preserving pancreaticoduodenectomy. Histopathological examination showed that the tumor had grown within a hollow structure, was contiguous with a duodenal diverticulum, and had partially invaded the pancreas. Immunohistochemistry results were as follows:CK7 negative, CK20 positive, CD10 negative, CDX2 positive, MUC1 negative, MUC2 positive, MUC5AC negative, and MUC6 negative. The tumor was diagnosed as duodenal carcinoma from the duodenal diverticulum. Preoperative imaging showed that the tumor was located in the head of the pancreas and was compressing the common bile duct, thus making it appear like pancreatic cancer. To the best of our knowledge, this is the second report of a case of duodenal carcinoma from a duodenal diverticulum mimicking pancreatic carcinoma. |
Keywords | duodenal carcinoma duodenal diverticulum pancreatic carcinoma |
Amo Type | Case Report |
Publication Title | Acta Medica Okayama |
Published Date | 2012-10 |
Volume | volume66 |
Issue | issue5 |
Publisher | Okayama University Medical School |
Start Page | 423 |
End Page | 427 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
Copyright Holders | CopyrightⒸ 2012 by Okayama University Medical School |
File Version | publisher |
Refereed | True |
PubMed ID | 23093061 |
Web of Science KeyUT | 000310253900007 |
JaLCDOI | 10.18926/AMO/46629 |
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FullText URL | 65_3_179.pdf |
Author | Teramen, Hirotake| Tsukuda, Kazunori| Tanaka, Norimitsu| Ueno, Tsuyoshi| Kubo, Takafumi| Ando, Midori| Soh, Junichi| Asano, Hiroaki| Pass, Harvery I.| Toyooka, Shinichi| Miyoshi, Shinichiro| |
Abstract | Suppression of p21 has been implicated in the genesis and progression of many human malignancies. DNA methylation is an important mechanism of gene silencing in human malignancies. In this study, we examined the expression status and aberrant methylaion of p21 in lung cancers and malignant pleural mesotheliomas (MPM). We used 12 small cell lung cancer (SCLC) cell lines, 13 non-small cell lung cancer (NSCLC) cell lines, 50 primary NSCLCs, 6 MPM cell lines and 10 primary MPMs. The expression and methylation of p21 was examined by reverse transcription-PCR (RT-PCR), Western blotting and methylation-specific PCR (MSP) assay. Loss of p21 protein expression was observed in 7 SCLC cell lines (58.3%), 5 NSCLC cell lines (38.5%) and 3 MPM cell lines (50%) while mRNA expression was lost in 2 SCLC cell lines (16.7%), 2 NSCLC cell lines (15.4%) and none of the MPM cell lines. Aberrant methylation of p21 was found in 8.3% of SCLC cell lines, 30.2% of NSCLCs and 6.3% of MPMs. Among primary NSCLCs, methylation in adenocarcinomas was significantly more frequent than in squamous cell carcinomas. Loss of p21 expression was frequently observed in lung cancers and MPMs and aberrant methylation was one of the mechanisms of suppression of p21, especially in NSCLCs. |
Keywords | p21 methylation lung cancer mesothelioma |
Amo Type | Original Article |
Publication Title | Acta Medica Okayama |
Published Date | 2011-06 |
Volume | volume65 |
Issue | issue3 |
Publisher | Okayama University Medical School |
Start Page | 179 |
End Page | 184 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
Copyright Holders | CopyrightⒸ 2011 by Okayama University Medical School |
File Version | publisher |
Refereed | True |
PubMed ID | 21709715 |
Web of Science KeyUT | 000292017500004 |
FullText URL | fulltext.pdf |
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Author | Yano, Masaaki| Ouchida, Mamoru| Shigematsu, Hisayuki| Tanaka, Noriyoshi| Ichimura, Koichi| Kobayashi, Kazuyasu| Inaki, Yasuhiko| Toyooka, Shinichi| Tsukuda, Kazunori| Shimizu, Nobuyoshi| Shimizu, Kenji| |
Keywords | alternative splicing HELLS loss of heterozygosity lung cancer SMARCA6 |
Note | Digital Object Identifer:10.1002/ijc.20407 Published with permission from the copyright holder. This is the author's copy, as published in the Journal of International Journal of Cancer, October 2004, Volume 112, Issue 1, Pages 8-13. Publisher URL:http://dx.doi.org/10.1002/ijc.20407 Direct access to Thomson Web of Science record Copyright © 2004 Wiley-Liss, Inc. All rights reserved.| |
Published Date | 2004-10-20 |
Publication Title | International Journal of Cancer |
Volume | volume112 |
Issue | issue1 |
Publisher | Wiley-Liss, Inc. |
Start Page | 8 |
End Page | 13 |
ISSN | 0020-7136 |
NCID | AA00680002 |
Content Type | Journal Article |
language | English |
OAI-PMH Set | 岡山大学 |
Copyright Holders | Wiley-Liss, Inc. |
File Version | author |
PubMed ID | 15305370 |
DOI | 10.1002/ijc.20407 |
Web of Science KeyUT | 000223939100002 |
JaLCDOI | 10.18926/AMO/32909 |
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FullText URL | fulltext.pdf |
Author | Tsukuda, Kazunori| Ikeda, Eiji| Takagi, Shoji| Miyake, Takayoshi| Muraoka, Takayuki| Watanabe, Keitaro| Hirai, Ryuji| Moriyama, Shigeharu| Nawa, Sugato| Kunitomo, Tadayoshi| Tsuji, Hisashi| |
Abstract | Gastrointestinal stromal tumors (GISTs) have been reported to occasionally occur in patients with neurofi bromatosis type 1 (NF-1), and many cases have had multiple lesions predominantly involving the small intestine. We report herein a case of multiple GISTs associated with NF-1 from whom laparoscopic surgery was benefi cial. In a 79-year-old female admitted with anemia and melena, the abdominal computed tomography revealed a tumor arising from the small intestine. Laparoscopic surgery was performed, and another small tumor was revealed during laparoscopic observation. Extracorporeal partial and wedge resection of the small intestine were undertaken. Both lesions were diagnosed as typical GISTs of low risk. Laparoscopic surgery would be useful for examination and a minimally invasive approach to tumors of the small intestine, especially on cases with the possibility of multiple tumors. |
Keywords | gastrointestinal stromal tumor neurofi bromatosis type 1 laparoscopic surgery |
Amo Type | Case Report |
Publication Title | Acta Medica Okayama |
Published Date | 2007-02 |
Volume | volume61 |
Issue | issue1 |
Publisher | Okayama University Medical School |
Start Page | 47 |
End Page | 50 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
File Version | publisher |
Refereed | True |
PubMed ID | 17332842 |
Web of Science KeyUT | 000244432400007 |
JaLCDOI | 10.18926/AMO/32891 |
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FullText URL | fulltext.pdf |
Author | Yuasa, Ichiro| Tsukuda, Kazunori| Hirai, Ryuji| Ota, Tetsuya| Murakami, Masakazu| Naito, Minoru| Doihara, Hiroyoshi| Date, Hiroshi| Shimizu, Nobuyoshi| |
Abstract | Resection is the only curative treatment for liver metastasis of colorectal cancers. Despite the supreme regenerative potential of the liver, major hepatectomy sometimes leads to liver failure, and the limitation of resectable liver volumes makes advanced tumors inoperable. This study was attempted to promote liver regeneration using hepatocyte growth factor (HGF) gene transfection by venous-administered adenovirus and to improve the survival of rats after massive hepatectomy. The adenovirus that encodes HGF was administered to rats before 85%-hepatectomy. The administration of HGF gene improved the survival of rats after massive hepatectomy, while the administration of control adenovirus deteriorated their survival. Gene transfection of HGF showed up-regulation of serum HGF, stimulation of hepatocellular proliferation and rapid liver regeneration. Moreover, HGF administration reduced apoptosis of hepatocytes. The administration of HGF gene prevented liver dysfunction after major hepatectomy and may be a new assist for surgery. |
Keywords | gene therapy hepatectomy HGF adenoviral vector |
Amo Type | Original Article |
Publication Title | Acta Medica Okayama |
Published Date | 2007-04 |
Volume | volume61 |
Issue | issue2 |
Publisher | Okayama University Medical School |
Start Page | 81 |
End Page | 88 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
File Version | publisher |
Refereed | True |
PubMed ID | 17471308 |
Web of Science KeyUT | 000245875600005 |
JaLCDOI | 10.18926/AMO/32112 |
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FullText URL | fulltext.pdf |
Author | Ota, Tetsuya| Hirai, Ryuji| Tsukuda, Kazunori| Murakami, Masakazu| Naitou, Minoru| Shimizu, Nobuyoshi| |
Abstract | We report a case requiring biliary reconstruction with right hepatic lobectomy due to biliary strictures caused by continuous cholangitis after laparoscopic bile duct injury. The patient, a 55-year-old woman, underwent laparoscopic cholecystectomy for cholelithiasis at another hospital. Although a bile leakage from the intraabdominal drain was observed several days after the operation, the patient was not given adequate treatment to stop the leakage. Two months after the initial laparoscopic cholecystectomy, she was referred to our hospital. Endoscopic retrograde cholangiopancreatography (ERCP) showed complete obstruction of the common hepatic duct, which was caused by clipping during laparoscopic cholecystectomy. Cholangiography from percutaneous transhepatic biliary drainage (PTBD) catheters revealed that sections of the secondary branches of the right intrahepatic bile duct had become constricted due to persistent cholangitis. Fortunately, the left hepatic duct was judged to be normal by imaging. Therefore, we elected to perform a right hepatic lobectomy and left hepaticojejunostomy, because we felt that performing a hepaticojejunostomy without hepatic resection would put the patient at risk of continuing to suffer from cholangitis. The patient was discharged on the 55 th postoperative day, and, 5 years after reconstructive surgery, is healthy and has remained free from biliary strictures in the remnant liver. Appropriate decision-making is essential in the treatment of biliary injury after laparoscopic cholecystectomy. Surgeons should not hesitate to perform biliary reconstruction with hepatic resection to reduce the risk of cholangitis or biliary strictures of the remnant liver. More importantly, preoperative clear imaging of the biliary tree and suitable management of any biliary injury which might occur are necessary to avoid having to perform reconstructive surgery. |
Keywords | ?biliary injury laparoscopic cholecystectomy hepatic resection |
Amo Type | Article |
Publication Title | Acta Medica Okayama |
Published Date | 2004-06 |
Volume | volume58 |
Issue | issue3 |
Publisher | Okayama University Medical School |
Start Page | 163 |
End Page | 167 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
File Version | publisher |
Refereed | True |
PubMed ID | 15471439 |
Web of Science KeyUT | 000222273300008 |
JaLCDOI | 10.18926/AMO/31828 |
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FullText URL | fulltext.pdf |
Author | Tsukuda, Kazunori| Tsuji, Hisashi| Kunitomo, Tadayoshi| Aokage, Keiju| Miyake, Takayoshi| Nakahara, Saki| Masuda, Hiroko| |
Abstract | Breast cancer with cartilaginous and/or osseous metaplasia is a type of metaplastic carcinomas and is a rare disease. We report the case of a 49 year-old female who underwent right mastectomy for a large breast tumor. Histological examinations revealed a mixed tumor with both stromal and epithelial elements;the stroma showed poor differentiated spindle-shape and multiform cells with a massive osseous matrix, and atypical epithelial cells, which mainly existed on the surface of the cysts, showed nucleic atypia. The tumor was diagnosed as a malignant phyllodes tumor with osteosarcomatous differentiation;it was not identified as a metaplastic carcinoma because of the lack of proof of a cancerous component. Two years after a mastectomy, swelling of the axillary lymph nodes was found and a biopsy was performed. Histological findings for the lymph node indicated a metastasis of the invasive ductal carcinoma. The primary tumor was re-examined and was considered to be the origin of the lymph nodal metastasis. Lymph nodal metastasis of cancer proved that the primary tumor had cancerous potential, and the pathological diagnosis was altered to a breast cancer with cartilaginous and/or osseous metaplasia. |
Keywords | breast cancer metaplastic cancer phyllodes tumor |
Amo Type | Case Report |
Publication Title | Acta Medica Okayama |
Published Date | 2009-12 |
Volume | volume63 |
Issue | issue6 |
Publisher | Okayama University Medical School |
Start Page | 367 |
End Page | 371 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
File Version | publisher |
Refereed | True |
PubMed ID | 20035293 |
Web of Science KeyUT | 000273145900008 |
JaLCDOI | 10.18926/AMO/30953 |
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FullText URL | fulltext.pdf |
Author | Tsukuda, Kazunori| Furutani, Shiro| Nakahara, Saki| Tada, Akihiro| Watanabe, Keitaro| Takagi, Shoji| Ikeda, Eiji| Hirai, Ryuji| Tsuji, Hisashi| |
Abstract | Abscess formation of the round ligament of the liver is very rare. We report a case of a 70-year-old female with abscess of the round ligament after an endoscopic papillotomy for choledocholithiasis. On the 21st day following papillotomy, abscess formation of the round ligament was found by ultrasonographic examination. Surgical treatment was performed because conservative therapy was not effective. The purulent fluid and necrotic tissue at the round ligament were completely removed. Cultures obtained from the abscess grew Staphylococcus epidermidis, but the mechanism of abscess formation in this case remains unclear. |
Keywords | round ligament of the liver abscess |
Amo Type | Case Report |
Publication Title | Acta Medica Okayama |
Published Date | 2008-12 |
Volume | volume62 |
Issue | issue6 |
Publisher | Okayama University Medical School |
Start Page | 411 |
End Page | 413 |
ISSN | 0386-300X |
NCID | AA00508441 |
Content Type | Journal Article |
language | English |
File Version | publisher |
Refereed | True |
Web of Science KeyUT | 000262025000008 |
Author | 佃 和憲| |
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Published Date | 2001-03-25 |
Publication Title | |
Content Type | Thesis or Dissertation |