| ID | 57519 |
| フルテキストURL | |
| 著者 |
Zhao, Lei
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
Le Thi Phuong
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
Mai Thanh Luan
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
Aprilia Nur Fitrianti
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
Matsui, Hidenori
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
Nakagami, Hirofumi
RIKEN Center for Sustainable Resource Science
Noutoshi, Yoshiteru
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
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Yamamoto, Mikihiro
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
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Ichinose, Yuki
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
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Shiraishi, Tomonori
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
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Toyoda, Kazuhiro
Laboratory of Plant Pathology and Genetic Engineering, Graduate School of Environmental and Life ScienceOkayama University
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| 抄録 | PRX34 mediates the oxidative burst in Arabidopsis. Here we characterized two additional Arabidopsis prx34 null mutants (prx34-2, prx34-3), besides the well-studied prx34-1. Due to a decrease in corresponding peroxidase, the activity that generates reactive oxygen species (ROS) was significantly lower in cell wall extracts of prx34-2 and prx34-3 plants. Consistently, the prx34-2 and prx34-3 exhibited reduced accumulation both of ROS and callose in Flg22-elicitor-treated leaves, leading to enhanced susceptibility to bacterial and fungal pathogens. In contrast, ectopic expression of PRX34 in the wild type caused enhanced resistance. PRX34 is thus a component for disease resistance in Arabidopsis.
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| キーワード | Apoplastic oxidative burst
Arabidopsis
Cell wall
Class III peroxidase
PRX34
Reactive oxygen species (ROS)
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| 備考 | This fulltext will be available in Jun 2020
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| 発行日 | 2019-06-07
|
| 出版物タイトル |
Journal of General Plant Pathology
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| 巻 | 85巻
|
| 号 | 6号
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| 出版者 | Springer
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| 開始ページ | 405
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| 終了ページ | 412
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| ISSN | 1345-2630
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| NCID | AA11449093
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| 資料タイプ |
学術雑誌論文
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| 言語 |
英語
|
| OAI-PMH Set |
岡山大学
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| 論文のバージョン | author
|
| DOI | |
| Web of Science KeyUT | |
| 関連URL | isVersionOf https://doi.org/10.1007/s10327-019-00863-9
|
| Citation | Zhao, L., Phuong, L.T., Luan, M.T. et al. J Gen Plant Pathol (2019) 85: 405. https://doi.org/10.1007/s10327-019-00863-9
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| 助成機関名 |
日本学術振興会
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| 助成番号 | 18K05645
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