start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2024
dt-pub=20240325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=結合組織型肥満細胞はNoradrenalineを貯蔵、放出する
kn-title=Connective tissue mast cells store and release noradrenaline
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=OTANIYusuke
en-aut-sei=OTANI
en-aut-mei=Yusuke
kn-aut-name=大谷悠介
kn-aut-sei=大谷
kn-aut-mei=悠介
aut-affil-num=1
ORCID=

affil-num=1
en-affil=Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University
kn-affil=岡山大学大学院医歯薬学総合研究科
END

start-ver=1.4
cd-journal=joma
no-vol=135
cd-vols=
no-issue=3
article-no=
start-page=174
end-page=174
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2023
dt-pub=20231201
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=The 55th Annual Meeting of the Japanese Society for Matrix Biology and Medicine
kn-title=第55回日本結合組織学会学術大会開催報告
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=OohashiToshitaka
en-aut-sei=Oohashi
en-aut-mei=Toshitaka
kn-aut-name=大橋俊孝
kn-aut-sei=大橋
kn-aut-mei=俊孝
aut-affil-num=1
ORCID=

affil-num=1
en-affil=Department of Molecular Biology and Biochemistry, Faculty of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University
kn-affil=岡山大学学術研究院医歯薬学域　分子医化学
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2010
dt-pub=20101231
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=圧負荷の心臓モデルにおける結合組織成長因子の誘導はAT1受容体阻害薬であるオルメサルタンにより改善された
kn-title=Induction of connective tissue growth factor in the pressure-overloaded heart was ameliorated by angiotensin �U type 1 receptor blocker olmesartan
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=IwamotoMutsumi
en-aut-sei=Iwamoto
en-aut-mei=Mutsumi
kn-aut-name=岩本睦
kn-aut-sei=岩本
kn-aut-mei=睦
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2009
dt-pub=20090325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=CCNファミリー2/結合組織成長因子は軟骨細胞の増殖と分化を調節し、シグナルコンダクターとしてBMPシグナル伝達経路を制御する
kn-title=CCN family 2/connective tissue growth factor modulates BMP signaling as a signal conductor, which action regulates the proliferation and differentiation of chondrocytes
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=MaedaAzusa
en-aut-sei=Maeda
en-aut-mei=Azusa
kn-aut-name=前田あずさ
kn-aut-sei=前田
kn-aut-mei=あずさ
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

start-ver=1.4
cd-journal=joma
no-vol=98
cd-vols=
no-issue=5-6
article-no=
start-page=475
end-page=485
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1986
dt-pub=19860630
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=Studies on the characteristics of human lung mast cells I. Purification and viability of human lung mast cells
kn-title=ヒト肺の肥満細胞に関する研究 第1編 ヒト肺肥満細胞の分離精製に関する検討
en-subtitle=
kn-subtitle=
en-abstract=In order to clarify the pathophysiology of allergic disorders, it is necessary to study the reactivity of mast cells. Recently, evidence that mast cells have specificities in species and organs has been reported. For use in an investigation of bronchial asthma, I developed a suitable technique for the purification of mast cells using normal lung tissue from patients with lung cancer. The purification steps were: �@ dispersion of lung fragments into a single cell suspension with an enzyme mixture (pronase-chymopapain and collagenase-elastase), �A centrifugation using a Percoll continuous gradient, and �B exclusion of adherent cells. As a result, 2.1±1.1×10(5) mast cells per gram of lung tissue in purities of 38.8±7.2% were obtained. Isolated mast cells proved functionally and morphologically stable by the following findings. The cells showed: �@ dye exclusion exceeding 95%, �A a histamine content of 4.3±0.6pg/mast cell, �B a histamine release of 47.9±11.2% induced by calcium ionophore A23187, and �C almost no damage on scanning electron microscopic observation. These results indicate that the present technique for the purification of human lung mast cells is comparatively simple and useful. The isolated mast cells can be applied to pathogenetic and therapeutic studies of bronchial asthma.
kn-abstract=19世紀後半にEhrlichによって見出された肥満細胞は,皮膚,肺,腸管をはじめ全身の臓器の結合組織血管周囲に多数分布しており,各種炎症巣において急性炎症期では減少し慢性炎症期では修復機転の続く限り増加するため,炎症反応の初期から修復に至る各時期に関与して外界の環境変化に対応する幅広い役割を担った細胞と考えられている.さらに1966年には石坂らのIgEの発見によってアレルギーの分野における肥満細胞の機能に関する研究が長足の進歩を遂げるに至っている1)〜3).このような肥満細胞の細胞化学,免疫薬理学的な研究においては材料の調整法の理由から主としてラットなど動物由来の肥満細胞が用いられている.また著者らはヒト肥満細胞の代りにヒト末梢血好塩基球を用い,その反応性をアレルギー反応の指標として一連の検討を行なっている4)〜9).しかし,肥満細胞と好塩基球の形態学的あるいは機能的な相違点10)〜14)については以前から指摘されており,また近年種属及び臓器間での肥満細胞のheterogeneityの存在が明らかにされてきた15),16)ことから,気管支喘息をはじめとするアレルギー性肺疾患の病態を解明する上で,各種動物由来の肥満細胞とヒト肺肥満細胞,さらにヒト末梢血好塩基球との相違点を比較検討し,ヒト肺肥満細胞の特異性を明らかにしておく必要があると思われる.以上の目的で従来からヒトの肺小片を用いた肥満細胞の研究17)〜19)が行なわれているが,試薬が組織中の肥満細胞に到達しにくく,かつ遊離された化学伝達物質も正確に定量できないという欠点がある.それを補なうために1972年にGouldらが家兎の肺組織を種々の酵素で処理し単離肥満細胞を分離した20)のに始まり,ヒト肺では1976年にAustenらが同様の方法で分離を試み21),1982年Lichtensteinらはその方法を発展させて高率のヒト肺単離肥満細胞を得ることに成功している22),しかしながら以上の方法では細胞の回収率や繁雑な精製手段などの問題点が残されている.そこで著者はヒト肺肥満細胞の機能を検討する手段として,外科的切除肺から肥満細胞を分離精製する方法とviabilityについて種々検討し,細胞障害性が少なく回収率が良く,かつ比較的簡便な分離法を考案したので報告する.
en-copyright=
kn-copyright=

en-aut-name=MaedaMasanori
en-aut-sei=Maeda
en-aut-mei=Masanori
kn-aut-name=前田昌則
kn-aut-sei=前田
kn-aut-mei=昌則
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学医学部第二内科学教室
en-keyword=human lung mast cell
kn-keyword=human lung mast cell
en-keyword=purification
kn-keyword=purification
en-keyword=viability
kn-keyword=viability
en-keyword=microscopic observation
kn-keyword=microscopic observation
en-keyword=histamine
kn-keyword=histamine
END

start-ver=1.4
cd-journal=joma
no-vol=65
cd-vols=
no-issue=
article-no=
start-page=96
end-page=100
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1994
dt-pub=199409
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=A case report of mixed connective tissue disease.
kn-title=混合性結合組織病(MCTD)の1症例
en-subtitle=
kn-subtitle=
en-abstract=A 50-year-old woman, diagnosed as rheumatoid arthritis (RA) in 1991, had been treated with Lobenzarit disodium and non steroidal anti-inflammatory drugs (NSAIDs) before
administration at our hospital. Despite the treatment with these drugs, her symptoms did not improve. She was admitted to our hospital for her progressive bilateral elbow joint pain and Raynaud's phenomenon. Although abnormal values of laboratory examinations such as an increased ESR (64mm/1hr), increased level of serum IgG (2854mg/?) and a positive RA test were shown, the clinical features of classical RA were not clear. There, she was diagnosed as mixed connective tissue disease (MCTD), because of high level of anti-RNP antibody in serum,
Raynaud's phenomenom, multiple arthritis and constrictive ventilatory disturbance. Her symptoms and the results of labolatory examinations were clearly improved by glucocorticoid therapy.
kn-abstract=症例は50歳女性で,1991年頃より両肘関節の疼痛とレイノー現象を自覚するようになり前医にて慢性関節リウマチ(Rheumatoid arthritis, 以下RAと略)との診断を受け加療受けるも症状の改善なく,当科を受診した｡典型的な慢性関節リウマチの症状に乏しく,他の膠原病の存在を疑って検索したところ,抗RNP抗体陽性等の所見から,混合性結合組織病(mixed connective tissue disease, 以下MCTDと略)と診断した｡MCTDは初診時に慢性関節リウマチと誤診される例が多く,慢性関節リウマチに非典型的な症状を伴う場合は,MCTDが鑑別診断として重要と考えられた。
en-copyright=
kn-copyright=

en-aut-name=TakataIchiro
en-aut-sei=Takata
en-aut-mei=Ichiro
kn-aut-name=高田一郎
kn-aut-sei=高田
kn-aut-mei=一郎
aut-affil-num=1
ORCID=

en-aut-name=KitaniHikaru
en-aut-sei=Kitani
en-aut-mei=Hikaru
kn-aut-name=貴谷光
kn-aut-sei=貴谷
kn-aut-mei=光
aut-affil-num=2
ORCID=

en-aut-name=MifuneTakashi
en-aut-sei=Mifune
en-aut-mei=Takashi
kn-aut-name=御舩尚志
kn-aut-sei=御舩
kn-aut-mei=尚志
aut-affil-num=3
ORCID=

en-aut-name=MitsunobuFumihiro
en-aut-sei=Mitsunobu
en-aut-mei=Fumihiro
kn-aut-name=光延文裕
kn-aut-sei=光延
kn-aut-mei=文裕
aut-affil-num=4
ORCID=

en-aut-name=KajimotoKazuhiro
en-aut-sei=Kajimoto
en-aut-mei=Kazuhiro
kn-aut-name=梶本和宏
kn-aut-sei=梶本
kn-aut-mei=和宏
aut-affil-num=5
ORCID=

en-aut-name=MatsumuraTadashi
en-aut-sei=Matsumura
en-aut-mei=Tadashi
kn-aut-name=松村正
kn-aut-sei=松村
kn-aut-mei=正
aut-affil-num=6
ORCID=

en-aut-name=YokotaSatoshi
en-aut-sei=Yokota
en-aut-mei=Satoshi
kn-aut-name=横田聡
kn-aut-sei=横田
kn-aut-mei=聡
aut-affil-num=7
ORCID=

en-aut-name=TanizakiYoshiro
en-aut-sei=Tanizaki
en-aut-mei=Yoshiro
kn-aut-name=谷崎勝朗
kn-aut-sei=谷崎
kn-aut-mei=勝朗
aut-affil-num=8
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学医学部附属病院三朝分院内科

affil-num=2
en-affil=
kn-affil=岡山大学医学部附属病院三朝分院内科

affil-num=3
en-affil=
kn-affil=岡山大学医学部附属病院三朝分院内科

affil-num=4
en-affil=
kn-affil=岡山大学医学部附属病院三朝分院内科

affil-num=5
en-affil=
kn-affil=岡山大学医学部附属病院三朝分院内科

affil-num=6
en-affil=
kn-affil=岡山大学医学部附属病院三朝分院内科

affil-num=7
en-affil=
kn-affil=岡山大学医学部附属病院三朝分院内科

affil-num=8
en-affil=
kn-affil=岡山大学医学部附属病院三朝分院内科
en-keyword=混合性結合組織病 (MCTD)
kn-keyword=混合性結合組織病 (MCTD)
en-keyword=慢性関節リウマチ (Rheumatoid arthritis)
kn-keyword=慢性関節リウマチ (Rheumatoid arthritis)
en-keyword=抗RNP抗体 (Anti-RNP antibody)
kn-keyword=抗RNP抗体 (Anti-RNP antibody)
END

start-ver=1.4
cd-journal=joma
no-vol=118
cd-vols=
no-issue=2
article-no=
start-page=131
end-page=137
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060901
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=
kn-title=眼科領域のアレルギー性疾患とアトピー性皮膚炎の眼合併症
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=アレルギー反応の4型, 1型 (lgE依存型反応), 1型 (細胞障害型反応), III型 (免疫複合体型反応), IV型 (細胞免疫型反応) を眼科疾患に当てはめると, I型はアレルギー性結膜疾患, II型は眼類天庖瘡やモーレン (Mooren) 角膜潰瘍などの自己免疫疾患, III型はぶどう球菌アレルギーによる周辺部角膜浸潤, IV型は角膜や結膜ブリクテン, 接触性皮膚炎による眼瞼炎, となる. その他, アレルギー関連疾患として, アトピー性皮膚炎がある. アトピー性皮膚炎は, 小児期の疾患であり, 通常, 皮膚は乾燥している. しかし日本では, 思春期から青年期, さらには成人になってから発症する型がある. この型は, 顔面におこることが多く, しかも皮膚は乾燥しておらず, 湿潤している. アトピー性白内障は, 顔面, 特に眼瞼に皮膚炎がある場合に見られる. 問診すると, 特に就寝時に眼瞼をたたく癖がある. アトピー性網膜剥離は, 通常, 白内障がある眼に起こっている. 硝子体手術を行うと, 硝子体基底部の綿状のきたない混濁がみられ, その近辺に鋸状縁断裂, 毛様体無色素上皮剥離や上皮裂孔がある. 一方, アトピー性白内障がある眼では, 前房水中のフレア値 (蛋白濃度) が上昇している. 以上の事実から, アトピー性皮膚炎の炎症が前眼部に波及し, それに眼瞼をたたくという外傷が加わって白内障や網膜剥離がおこると考えられる.
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=松尾俊彦
kn-aut-sei=松尾
kn-aut-mei=俊彦
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学大学院医歯薬学総合研究科 眼科学
en-keyword=アレルギー性結膜炎
kn-keyword=アレルギー性結膜炎
en-keyword=春季カタル
kn-keyword=春季カタル
en-keyword=巨大乳頭結膜炎
kn-keyword=巨大乳頭結膜炎
en-keyword=アトピー性皮膚炎
kn-keyword=アトピー性皮膚炎
en-keyword=アトピー性網膜剥離
kn-keyword=アトピー性網膜剥離
en-keyword=アトピー性白内障
kn-keyword=アトピー性白内障
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2008
dt-pub=20080630
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=プロコラーゲンC末端プロテナーゼ　エンハンサー1(PCPE-1)は、結合組織上にてβ2マイクログロブリンと相互作用し、β2マイクログロブリン由来アミロイド線維形成を助ける可能性がある
kn-title=Procollagen C-proteinase enhancer-1 (PCPE-1) interacts with β2-microglobulin (β2-m) and may help initiate β2-m amyloid fibril formation in connective tissues
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=MorimotoHisanori
en-aut-sei=Morimoto
en-aut-mei=Hisanori
kn-aut-name=森本尚孝
kn-aut-sei=森本
kn-aut-mei=尚孝
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

start-ver=1.4
cd-journal=joma
no-vol=61
cd-vols=
no-issue=
article-no=
start-page=98
end-page=113
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1990
dt-pub=199009
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=New trend of the development of vascular prostheses
kn-title=人工血管開発における研究の動向
en-subtitle=
kn-subtitle=
en-abstract=Vascular prostheses have been used in the treatments of various vascular diseases. We expect much from their contribution in the field of further fine vascular surgery. In this communication, new trend of the development of them with our new ideas in our recent
research works were described. 1. Healing process of fabric vascular prostheses. Neointima formation of the vascular prosthesis implanted in the descending thoracic
aortae of experimental animals were described in detail. This showed a standard behaviour of various cells which contributed the construction of a new arterial wall.
2. Problems of vascular prostheses which were used in clinic. Several problems such as a bleeding from the graft wall, aneurysmal dilatation, and degenerative changes of the neointimae were explained in detail. These problems were related with the structure of each vascular prosthesis. 3. Vascular prostheses developing with new
ideas. Several vascular prostheses developing today were described as follows. (a) Fabric prosthesis with high healing ability. A prosthesis made of ultra-fine polyester
fibers which can accerelate cell migration and proliferation inside the prosthetic wall was introduced. The neointima in this prosthesis was constructed very rapidly compared with that of the prostheses made of polyester fiber ordinally used. (b) Antithrombogenic small
diameter vascular prostheses. Large caliber vascular prostheses have been used very safely in clinic, however, small diameter ones were seldom used due to its low patency rate by their occlusion. The grafts should have an antithrombogenic property to prevent the thrombus formation. Recently, some technologies to give antithrombogenic property to the vascular prostheses were developed. They were a heparin slow release technique,
antithrombogenic segmented polyurethane, highly hydrous surface, heparinized collagen tube, etc. They were applied for the development of small diameter vascular grafts and showed good results in some animal experiments. (c) Growable vascular graft. A vascular graft which can grow with the growth of its recipient baby was demonstrated. The animal study showed as expected growth and stop growing
at the expected time. (d) Vascular graft for aorto-coronary bypass surgery. A small diameter antithrombogenic, and flexible vascular grafts were explained. They were developed along our own ideas. This showed high patency rate m animal experiments. (e)Endothelial cell seeding techniques. In human, endothelialization m the vascular grafts is delayed and the inner surface was thrombogenic for a long period of time. To accerelate the endothelialization, cell seeding
technique have been investigated in the last decade. We developed a new technology to transplant autologous cells in the peripheral vein tissue for this purpose. This was very reliable and simple. It was applicable in any hospital without any special technique and facility. Peripheral venous tissue fragments were transplanted into the fabric vascular prosthetic wall. Endothelial cells
and smooth muscle cells migrated and proliferated from the tissue fragments. The neointima was constructed very rapidly with this active cell migrations.
kn-abstract=人工血管の開発研究における動向について，我々の行ってきた研究をまじえて世界の動きを解析した。近年の砺究の流れは過去1世紀にわたる試行錯誤の上にあって，さらに大きく揺れ動いている。人工血管の内面に必要な抗血栓性の獲得においても，設計者によって考えを異にし，米国では，人工心臓に用いる抗血栓性合性高分子材料をもってそれに当てようとしているものが多い。我々はあくまで宿主のもつ諸機能を引き出し，自然の動きの一つとしての治癒を無理なく導いて，内皮細胞によって内面を覆わせる方法を採用している。そのいくつかの例として，超極細ポリエステル繊維製人工血管，結合組織管，バイオマトリックス人工血管，ヘパリン化生体組織由来人工血管，成長できる人工血管などについて解説した。
en-copyright=
kn-copyright=

en-aut-name=NoishikiYasuharu
en-aut-sei=Noishiki
en-aut-mei=Yasuharu
kn-aut-name=野一色泰晴
kn-aut-sei=野一色
kn-aut-mei=泰晴
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学医学部附属環境病態研究施設リハビリテーション外科学分野
en-keyword=人工血管 (Vascular prosthesis)
kn-keyword=人工血管 (Vascular prosthesis)
en-keyword=新生内膜 (Neointima)
kn-keyword=新生内膜 (Neointima)
en-keyword=内皮細胞 (Endothelial cells)
kn-keyword=内皮細胞 (Endothelial cells)
en-keyword=抗血栓性 (Antithrombogenicity)
kn-keyword=抗血栓性 (Antithrombogenicity)
END

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cd-journal=joma
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dt-pub-year=2006
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en-title=
kn-title=マウス実験的歯の移動における結合組織成長因子CTGFとアポトーシスの誘導
en-subtitle=
kn-subtitle=
en-abstract=
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en-aut-name=SakaiYuichi
en-aut-sei=Sakai
en-aut-mei=Yuichi
kn-aut-name=酒井雄一
kn-aut-sei=酒井
kn-aut-mei=雄一
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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kn-subject=
en-title=軟骨細胞におけるマクロファージコロニー刺激因子(M-CSF)と結合組織成長因子(CTGF/CCN2)の協調的作用―関節軟骨代謝における組織再生的役割の可能性―
kn-title=Collaborative action of M-CSF and CTGF/CCN2 in articular chondrocytes : Possible regenerative roles in articular cartilage metabolism
en-subtitle=
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en-aut-name=NakaoKyoji
en-aut-sei=Nakao
en-aut-mei=Kyoji
kn-aut-name=中尾匡志
kn-aut-sei=中尾
kn-aut-mei=匡志
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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cd-journal=joma
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dt-pub-year=2005
dt-pub=20050325
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kn-subject=
en-title=
kn-title=構成モジュール特異的抗体による結合組織成長因子CTGF/Hcs24の定量と機能解析
en-subtitle=
kn-subtitle=
en-abstract=
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kn-copyright=

en-aut-name=MinatoMasanao
en-aut-sei=Minato
en-aut-mei=Masanao
kn-aut-name=湊雅直
kn-aut-sei=湊
kn-aut-mei=雅直
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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dt-pub-year=2002
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kn-subject=
en-title=
kn-title=ラット下顎枝骨折治癒過程における結合組織成長因子CTGFの経時的発現
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
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en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=福永智広
kn-aut-sei=福永
kn-aut-mei=智広
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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kn-subject=
en-title=1.(チロシンキナーゼ型レセプターErbB4遺伝子の軟骨における発現:結合組織成長因子(CTGF/Hcs24)との関連と軟骨細胞での分布) 2.マクロフｧージコロニー刺激因子受容体遺伝子(m-csfr/c-fms)の軟骨細胞における発現
kn-title=1.Tyrosine kinase-field name=
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
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kn-copyright=

en-aut-name=NawachiKumiko
en-aut-sei=Nawachi
en-aut-mei=Kumiko
kn-aut-name=縄稚久美子
kn-aut-sei=縄稚
kn-aut-mei=久美子
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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dt-pub-year=2000
dt-pub=20000325
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kn-subject=
en-title=
kn-title=結合組織成長因子CTGF/ Hcs24の歯根膜線維芽細胞の増殖,分化に与える影響
en-subtitle=
kn-subtitle=
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kn-abstract=
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en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=浅野将宏
kn-aut-sei=浅野
kn-aut-mei=将宏
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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dt-received=
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dt-pub-year=2003
dt-pub=20030325
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kn-subject=
en-title=結合組織成長因子(CTGF)の発現がもたらすヒト口腔扁平上皮癌細胞株の増殖抑制効果
kn-title=Suppressive effect of overexpressed connective tissue growth factor on tumor cell growth in a human oral squamous cell carcinoma derived cell line
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
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en-aut-name=MoritaniNorifumi
en-aut-sei=Moritani
en-aut-mei=Norifumi
kn-aut-name=森谷徳文
kn-aut-sei=森谷
kn-aut-mei=徳文
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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cd-journal=joma
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dt-pub-year=2003
dt-pub=20030325
dt-online=
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en-subject=
kn-subject=
en-title=1.ヒト軟骨細胞様細胞株HCS-2/8におけるヒト結合組織成長因子CTGF/Hcs24遺伝子の発現制御機構) 2.軟骨細胞様細胞において結合組織成長因子の遺伝子発現を増強する新規シスエレメント)
kn-title=1.Regulatory mechanism of human connective tissue growth factor(CTGF/Hcs24)gene expression in a human chondrocytic cell line, HCS-2/8 2.Anovel cis-element that enhances connective tissue growth factor gene expression in chondrocytic cells
en-subtitle=
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en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=江口傑徳
kn-aut-sei=江口
kn-aut-mei=傑徳
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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cd-journal=joma
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dt-pub-year=2003
dt-pub=20030325
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kn-subject=
en-title=低酸素誘導された結合組織成長因子はマトリクスメタロプロテアーゼと協調して血管新生を誘発する
kn-title=Connective tissue growth factor increased by hypoxia may initiate angiogenesis in collaboration with matrix metalloproteinases
en-subtitle=
kn-subtitle=
en-abstract=
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en-copyright=
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en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=近藤誠二
kn-aut-sei=近藤
kn-aut-mei=誠二
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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cd-journal=joma
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dt-pub-year=2001
dt-pub=20010325
dt-online=
en-article=
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kn-subject=
en-title=歯肉線維芽細胞はグルタミン酸脱炭酸酵素(GAD)65を発現する:結合組織における炎症がGAD抗体価に与える影響
kn-title=Human Fibroblasts Ubiquitously Express Glutamic Acid Decarboxylase 65(GAD 65): Possible Effects of Connective Tissue Inflammation on GAD Antibody Titer
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=河野隆幸
kn-aut-sei=河野
kn-aut-mei=隆幸
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
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article-no=
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dt-pub-year=2000
dt-pub=20001231
dt-online=
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kn-article=
en-subject=
kn-subject=
en-title=軟骨性腫瘍におけるCTGF(結合組織成長因子)の発現
kn-title=Expression of Connective Tissue Growth Factor in Cartilaginous Tumors
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=杓永俊彦
kn-aut-sei=杓永
kn-aut-mei=俊彦
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END

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cd-journal=joma
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dt-pub-year=1979
dt-pub=19791231
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kn-subject=
en-title=同種および異種結合組織管の代用血管としての応用
kn-title=Homologous and Heterologous Connective tissue tube for Vascular Substitute.
en-subtitle=
kn-subtitle=
en-abstract=
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en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=山根義久
kn-aut-sei=山根
kn-aut-mei=義久
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=岡山大学
END