We reported a 62-year-old man with malacoplakia of the prostate, and reviewed 49 cases of malacoplakia hitherto observed in Japan in which the lesions originated from the urogenital tract, except for one gastric case. E. Coli was emphasized as a possible causative agent for malacoplakia especially in the urogenital tract. The possible histiocytic origin of von Hansemann cells was stressed by demonstrating cytoplasmic processes and desmosomes in our prostatic case. An adjuvant use of cholinergic agents and ascorbic acid with chemotherapeutic agents was recommended for treating malacoplakia.
en-copyright= kn-copyright= en-aut-name=KumonHiromi en-aut-sei=Kumon en-aut-mei=Hiromi kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=FurukawaMasato en-aut-sei=Furukawa en-aut-mei=Masato kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= en-aut-name=TsugawaMasaya en-aut-sei=Tsugawa en-aut-mei=Masaya kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=3 ORCID= en-aut-name=MatsumuraYosuke en-aut-sei=Matsumura en-aut-mei=Yosuke kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=4 ORCID= en-aut-name=OhmoriHiroyuki en-aut-sei=Ohmori en-aut-mei=Hiroyuki kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=5 ORCID= en-aut-name=TanakaToshio en-aut-sei=Tanaka en-aut-mei=Toshio kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=6 ORCID= affil-num=1 en-affil= kn-affil=Okayama University affil-num=2 en-affil= kn-affil=Okayama University affil-num=3 en-affil= kn-affil=Okayama University affil-num=4 en-affil= kn-affil=Okayama University affil-num=5 en-affil= kn-affil=Okayama University affil-num=6 en-affil= kn-affil=Okayama University en-keyword=malacoplakia kn-keyword=malacoplakia en-keyword=prostate kn-keyword=prostate en-keyword=von Hansemann cell kn-keyword=von Hansemann cell END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=519 end-page=520 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=A simplified quantification method of complex-release activity using peroxidase as immune complex antigen. en-subtitle= kn-subtitle= en-abstract= kn-abstract=The complement-mediated solubilization of precipitable immune complexes (complex-release activity) in serum specimens was determined by a simplified method using peroxidase as an immune complex antigen. The results correlated well with the hemolytic activity via the classical complement pathway and that via the alternative complement pathway. This simplified method proved to be reliable and useful.
en-copyright= kn-copyright= en-aut-name=AmanoTetsuki en-aut-sei=Amano en-aut-mei=Tetsuki kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=AibaraYasushi en-aut-sei=Aibara en-aut-mei=Yasushi kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= en-aut-name=KuwajimaNorio en-aut-sei=Kuwajima en-aut-mei=Norio kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=3 ORCID= en-aut-name=SuzukiSinya en-aut-sei=Suzuki en-aut-mei=Sinya kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=4 ORCID= en-aut-name=OtaZensuke en-aut-sei=Ota en-aut-mei=Zensuke kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=5 ORCID= affil-num=1 en-affil= kn-affil=Okayama University affil-num=2 en-affil= kn-affil=Okayama University affil-num=3 en-affil= kn-affil=Okayama University affil-num=4 en-affil= kn-affil=Okayama University affil-num=5 en-affil= kn-affil=Okayama University en-keyword=solubilization of immune complex kn-keyword=solubilization of immune complex en-keyword=complement kn-keyword=complement en-keyword=peroxidase kn-keyword=peroxidase END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=525 end-page=527 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Effect of continued drinking on prognosis of alcoholic liver cirrhosis. en-subtitle= kn-subtitle= en-abstract= kn-abstract=The prognoses of patients with alcoholic liver cirrhosis were compared between those who continued to drink and those who stopped. Clinical criteria were strictly set so as to control other variables affecting the prognoses. Four-year survival was significantly higher in the patients who stopped drinking than in those who continued to drink. Continued drinking worsens the prognosis of patients with alcoholic liver cirrhosis.
en-copyright= kn-copyright= en-aut-name=KobayashiMichio en-aut-sei=Kobayashi en-aut-mei=Michio kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=WatanabeAkiharu en-aut-sei=Watanabe en-aut-mei=Akiharu kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= en-aut-name=NakatsukasaHarushige en-aut-sei=Nakatsukasa en-aut-mei=Harushige kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=3 ORCID= en-aut-name=FujiwaraMasachika en-aut-sei=Fujiwara en-aut-mei=Masachika kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=4 ORCID= en-aut-name=ShiotaTetsuya en-aut-sei=Shiota en-aut-mei=Tetsuya kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=5 ORCID= en-aut-name=TakeiNobuyuki en-aut-sei=Takei en-aut-mei=Nobuyuki kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=6 ORCID= en-aut-name=SakataTatsuro en-aut-sei=Sakata en-aut-mei=Tatsuro kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=7 ORCID= en-aut-name=YamauchiYasuhiko en-aut-sei=Yamauchi en-aut-mei=Yasuhiko kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=8 ORCID= en-aut-name=NagashimaHideo en-aut-sei=Nagashima en-aut-mei=Hideo kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=9 ORCID= affil-num=1 en-affil= kn-affil=Okayama University affil-num=2 en-affil= kn-affil=Okayama University affil-num=3 en-affil= kn-affil=Okayama University affil-num=4 en-affil= kn-affil=Okayama University affil-num=5 en-affil= kn-affil=Okayama University affil-num=6 en-affil= kn-affil=Okayama University affil-num=7 en-affil= kn-affil=Okayama University affil-num=8 en-affil= kn-affil=Okayama University affil-num=9 en-affil= kn-affil=Okayama University en-keyword=alcoholic liver cirrhosis kn-keyword=alcoholic liver cirrhosis en-keyword=prognosis kn-keyword=prognosis en-keyword=continued drinking kn-keyword=continued drinking en-keyword=stopped drinking kn-keyword=stopped drinking en-keyword=alcohol consumption kn-keyword=alcohol consumption END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=457 end-page=462 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Serum fucosyl transferase activity and serum fucose levels as diagnostic tools in malignancy. en-subtitle= kn-subtitle= en-abstract= kn-abstract=Glycoproteins play a significant role in neoplastic transformations. Both the levels of fucose and the activity of fucosyl transferase, which mediates the assembly of the oligosaccharide moieties of the glycoprotein chains, have been found to be elevated in neoplastic conditions. Since these elevations are common features of a variety of neoplastic cells, these two have been designated as non-specific markers of malignancy. In the present study, the fucose level and fucosyl transferase activity were determined in the sera of cancer patients and an attempt was made to establish a relationship between the two. It was found that both the fucose levels and fucosyl transferase activities showed considerable elevation in the five cancer groups studied, establishing them as useful diagnostic parameters. However, it was also observed that the rate of increased fucosyl transferase activity was not fully reflected in the resulting serum fucose levels in a few cases.
en-copyright= kn-copyright= en-aut-name=SenUmi en-aut-sei=Sen en-aut-mei=Umi kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=GuhaSubhas en-aut-sei=Guha en-aut-mei=Subhas kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= en-aut-name=ChowdhuryJ Roy en-aut-sei=Chowdhury en-aut-mei=J Roy kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=3 ORCID= affil-num=1 en-affil= kn-affil=Chittaranjan National Cancer Research Center affil-num=2 en-affil= kn-affil=Chittaranjan National Cancer Research Center affil-num=3 en-affil= kn-affil=Chittaranjan National Cancer Research Center en-keyword=glycoprotein- fucose- fucosyl transferase kn-keyword=glycoprotein- fucose- fucosyl transferase en-keyword=non specific marker kn-keyword=non specific marker END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=483 end-page=491 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Immunochemotherapy with levamisole for stage III gastric cancer patients. en-subtitle= kn-subtitle= en-abstract= kn-abstract=Levamisole (LMS) was given to stage III gastric cancer patients starting three days before gastrectomy, at a does of 150 mg/day for three consecutive days every other week. Survival rates of these patients were compared with those of stage III gastric cancer patients previously operated in our Department who had not received levamisole. The background factors of both groups were matched as closely as possible. Both groups were concomitantly treated with mitomycin C and FT-207. The survival rate of the LMS group was significantly higher than that of the control group when the tumor had a diameter of 4.0-8.0 cm, cancer cells infiltrated to the gastric serosa, there were metastases within the regional lymph nodes, cancer cells slightly invaded the venous capillaren and there was moderate infiltration of the stroma.
en-copyright= kn-copyright= en-aut-name=MiwaHiroaki en-aut-sei=Miwa en-aut-mei=Hiroaki kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=OritaKunzo en-aut-sei=Orita en-aut-mei=Kunzo kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= affil-num=1 en-affil= kn-affil=Okayama University affil-num=2 en-affil= kn-affil=Okayama University en-keyword=gastric cancer kn-keyword=gastric cancer en-keyword=immunochemotherapy kn-keyword=immunochemotherapy en-keyword=levamisole kn-keyword=levamisole en-keyword=survival rate kn-keyword=survival rate END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=471 end-page=481 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Active enhancement of rat cardiac allografts induced by donor specific semisoluble antigens. en-subtitle= kn-subtitle= en-abstract= kn-abstract=Active enhancement was induced in inbred rats with cardiac allografts using semisoluble antigens. The optimal time of antigen pretreatment and optimal dose of semisoluble antigens were examined. The presence of serum blocking factors in the sera of rats having had allografts for a long time was examined with a macrophage migration inhibition test and lymphocyte microcytotoxicity assay. Since the blocking factors of macrophage migration inhibition were increasing on the 7th day, that day was determined to be the optimal time of antigen pretreatment. The mean survival time (MST) of cardiac allografts in untreated rats was 17.2 +/- 7.5 days. Semisoluble antigens were administered at 2 mg/kg body weight 7 days before the graft, 4 mg/kg 7 days before the graft and 2 mg/kg divided over three days, 15, 8 and 1 day before the graft, and the MSTs of cardiac allografts of rats receiving these treatments were 71.2 +/- 39.9, 62.6 +/- 42.2 and 79.3 +/- 31.0 days, respectively. The MST in each group of the treated rats was significantly longer than that of the control group (p less than 0.01). Rejection of the allograft, however, was accelerated in a group treated with 8 mg/kg 7 days before the graft (MST: 8.4 +/- 3.2 days). Serum blocking factors were detected in the sera of approximately half of the rats having cardiac allografts which survived a long time.
en-copyright= kn-copyright= en-aut-name=FuchimotoSadanori en-aut-sei=Fuchimoto en-aut-mei=Sadanori kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= affil-num=1 en-affil= kn-affil=Okayama University en-keyword=active enhancement kn-keyword=active enhancement en-keyword=optimal time of antigen pretreatment kn-keyword=optimal time of antigen pretreatment en-keyword=serum blocking factor kn-keyword=serum blocking factor en-keyword=rat cardiac allograft kn-keyword=rat cardiac allograft END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=521 end-page=523 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Seropositivity of a blood recipient from a donor with positive adult T-cell leukemia-associated antigens. en-subtitle= kn-subtitle= en-abstract= kn-abstract=A blood recipient, aged 66, was found to have positive adult T-cell leukemia-associated antigens (ATLA), approximately half a year after a transfusion. The donor's ATLA-antibody titer was 1: 640. Routine screening of blood donors for ATLA antibody was proposed.
en-copyright= kn-copyright= en-aut-name=MiyamotoKanji en-aut-sei=Miyamoto en-aut-mei=Kanji kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=TomitaNorio en-aut-sei=Tomita en-aut-mei=Norio kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= en-aut-name=IshiiAkio en-aut-sei=Ishii en-aut-mei=Akio kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=3 ORCID= en-aut-name=NishizakiTakeshi en-aut-sei=Nishizaki en-aut-mei=Takeshi kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=4 ORCID= en-aut-name=KitajimaKo-ichi en-aut-sei=Kitajima en-aut-mei=Ko-ichi kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=5 ORCID= en-aut-name=TanakaToshio en-aut-sei=Tanaka en-aut-mei=Toshio kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=6 ORCID= affil-num=1 en-affil= kn-affil=Okayama Red Cross Blood Center affil-num=2 en-affil= kn-affil=Okayama Red Cross Blood Center affil-num=3 en-affil= kn-affil=Okayama Red Cross Blood Center affil-num=4 en-affil= kn-affil=Okayama Red Cross Blood Center affil-num=5 en-affil= kn-affil=Okayama University affil-num=6 en-affil= kn-affil=Okayama University en-keyword=blood trasfusion kn-keyword=blood trasfusion en-keyword=adult T-cell leukemia virus kn-keyword=adult T-cell leukemia virus en-keyword=adult T-cell leukemia kn-keyword=adult T-cell leukemia END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=463 end-page=470 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Glutathione metabolism and glucose 6-phosphate dehydrogenase activity in experimental liver injury. en-subtitle= kn-subtitle= en-abstract= kn-abstract=Increased activities of liver glucose-6-phosphate dehydrogenase (G6PD, EC 1.1.1.49) and 6-phosphogluconate dehydrogenase (6PGD, EC 1.1.1.44) in the pentose phosphate cycle were accompanied with a depletion of reduced glutathione (GSH) following an intragastric administration of carbon tetrachloride (CCl4) to rats. Oxidized glutathione (GSSG) also decreased remarkably, keeping the GSSG: GSH ratio constant. No significant alteration of glutathione reductase (EC 1.6.4.2.), glutathione peroxidase (EC 1.11.1.9) and malic enzyme (EC 1.1.1.40) activities in the supernatant and gamma-glutamyl transpeptidase (gamma-GTP, EC 2.3.2.2) activity in the homogenate of the injured liver were observed. Furthermore, no marked difference in the GSH-synthesizing activity was found between control and CCl4-intoxicated liver. An intraperitoneal injection of GSH produced a significant increase in liver GSH content in control rats but not in CCl4-treated rats; G6PD activity was not affected. Intraperitoneal injections of diethylmaleate resulted in continuously diminished levels of liver GSH without any alteration of liver G6PD activity. In vitro disappearance of GSH added to the liver homogenate from CCl4-treated rats occurred enzymatically and could not be prevented by the addition of a NADPH-generating system. The results suggest that increased G6PD activity in CCl4-injured liver does not play an important role in the maintenance of glutathione in the reduced form and that the decreased GSH content in the injured liver might be caused by enhanced GSH catabolism not due to gamma-GTP.
en-copyright= kn-copyright= en-aut-name=WatanabeAkiharu en-aut-sei=Watanabe en-aut-mei=Akiharu kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=NagashimaHideo en-aut-sei=Nagashima en-aut-mei=Hideo kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= affil-num=1 en-affil= kn-affil=Okayama University affil-num=2 en-affil= kn-affil=Okayama University en-keyword=G6PD kn-keyword=G6PD en-keyword=glutathione kn-keyword=glutathione en-keyword=GSH kn-keyword=GSH en-keyword=GSSG kn-keyword=GSSG en-keyword=CCL kn-keyword=CCL en-keyword=liver injury kn-keyword=liver injury en-keyword=diethylmaleate kn-keyword=diethylmaleate END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=529 end-page=533 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Type C virus particles produced in human T-cell lines derived from acute lymphoblastic leukemia and a leukemic T-lymphoid malignancy. en-subtitle= kn-subtitle= en-abstract= kn-abstract=Electron microscopy of four human T-cell lines revealed the production of type C virus particles in two T-cell lines: one derived from acute lymphoblastic leukemia and the other from a leukemic T-lymphoid malignancy. Virus particles isolated from these cells had reverse transcriptase activity and the major internal structural protein of 30,000 daltons (p30). The indirect immunofluorescence test of these virus-producing cells with sera of patients with adult T-cell leukemia (ATL) was negative. The data indicate that these retroviruses are different from adult T-cell leukemia virus (ATLV).
en-copyright= kn-copyright= en-aut-name=OdaTakuzo en-aut-sei=Oda en-aut-mei=Takuzo kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=WatanabeSekiko en-aut-sei=Watanabe en-aut-mei=Sekiko kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= en-aut-name=NakamuraTakashi en-aut-sei=Nakamura en-aut-mei=Takashi kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=3 ORCID= affil-num=1 en-affil= kn-affil=Okayama University affil-num=2 en-affil= kn-affil=Okayama University affil-num=3 en-affil= kn-affil=Okayama University en-keyword=type C virus particles kn-keyword=type C virus particles en-keyword=human T-cell lines kn-keyword=human T-cell lines en-keyword=electron microscopy kn-keyword=electron microscopy en-keyword=virion proteins kn-keyword=virion proteins en-keyword=immunofluorescence test kn-keyword=immunofluorescence test END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=511 end-page=517 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Failure of droperidol and ketamine to influence cerebrospinal fluid production in the dog. en-subtitle= kn-subtitle= en-abstract= kn-abstract=Using the ventriculo-cisternal perfusion method, the effects of droperidol and ketamine hydrochloride on cerebrospinal fluid (CSF) production were studied in dogs. Neither droperidol (0.25 mg/kg, IV) nor ketamine (3 mg/kg, IV) caused a statistically significant change in CSF production rate. Positive correlation between CSF production and corresponding cerebral perfusion pressure (CPP) was observed in the ketamine study, whose unfavorable effect on neurosurgical anaesthesia would be obvious. On the other hand droperidol (0.25 mg/kg, IV) tended to decrease CSF production. Droperidol alone or in combination with other analgesics such as fentanyl as currently used in neurosurgical anaesthesia appears to be an appropriate choice in patients with increased intracranial pressure.
en-copyright= kn-copyright= en-aut-name=IkedaShigemasa en-aut-sei=Ikeda en-aut-mei=Shigemasa kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=SchwelssJohn F en-aut-sei=Schwelss en-aut-mei=John F kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= affil-num=1 en-affil= kn-affil=Saint Louis University affil-num=2 en-affil= kn-affil=Saint Louis University en-keyword=cerebrospinal fluid production kn-keyword=cerebrospinal fluid production en-keyword=ketamine kn-keyword=ketamine en-keyword=droperidol kn-keyword=droperidol END start-ver=1.4 cd-journal=joma no-vol=37 cd-vols= no-issue=6 article-no= start-page=503 end-page=510 dt-received= dt-revised= dt-accepted= dt-pub-year=1983 dt-pub=198312 dt-online= en-article= kn-article= en-subject= kn-subject= en-title= kn-title=Computed tomographic arteriography in the diagnosis of hepatocellular carcinoma. en-subtitle= kn-subtitle= en-abstract= kn-abstract=Computed tomographic arteriography (CTA) was performed in 30 patients with hepatocellular carcinoma (HCC). Detection of HCC by CTA was compared with that of conventional celiac or hepatic arteriography. CT scanning was performed immediately, 30 seconds and 1 min after an injection of 5 to 10 ml of contrast medium into the common or proper hepatic artery. Repeated infusions allowed whole liver sections to be visualized. HCC was localized in 28 of the 30 patients by conventional arteriography, with CTA detecting the masses in 27 of the 28 patients. CTA imaging presented the tumor mass in 1 of the 2 patients missed by arteriography. Conventional arteriography delineated the boundaries of HCC in 15 (50%) of the 30 patients. CTA clearly delineated the masses in 26 (87%) of the 30 patients including 11 patients in which the tumor borders were obscure by conventional arteriography. HCC lesions smaller than 1 cm in diameter were detected only by CTA in 6 (20%) of the patients. It was concluded that CTA is both useful and necessary in the demarcation of small HCC masses.
en-copyright= kn-copyright= en-aut-name=ItoToshio en-aut-sei=Ito en-aut-mei=Toshio kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=1 ORCID= en-aut-name=YamamotoHiroshi en-aut-sei=Yamamoto en-aut-mei=Hiroshi kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=2 ORCID= en-aut-name=ItoshimaTatsuya en-aut-sei=Itoshima en-aut-mei=Tatsuya kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=3 ORCID= en-aut-name=UkidaMinoru en-aut-sei=Ukida en-aut-mei=Minoru kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=4 ORCID= en-aut-name=OgawaHiromichi en-aut-sei=Ogawa en-aut-mei=Hiromichi kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=5 ORCID= en-aut-name=KitadaiMasahiro en-aut-sei=Kitadai en-aut-mei=Masahiro kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=6 ORCID= en-aut-name=HattoriShuzo en-aut-sei=Hattori en-aut-mei=Shuzo kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=7 ORCID= en-aut-name=MizutaniShigeki en-aut-sei=Mizutani en-aut-mei=Shigeki kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=8 ORCID= en-aut-name=KitaKeiji en-aut-sei=Kita en-aut-mei=Keiji kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=9 ORCID= en-aut-name=TanakaRyoji en-aut-sei=Tanaka en-aut-mei=Ryoji kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=10 ORCID= en-aut-name=YamauchiYasuhiko en-aut-sei=Yamauchi en-aut-mei=Yasuhiko kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=11 ORCID= en-aut-name=HashimotoKeiji en-aut-sei=Hashimoto en-aut-mei=Keiji kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=12 ORCID= en-aut-name=HayashiHidehiro en-aut-sei=Hayashi en-aut-mei=Hidehiro kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=13 ORCID= en-aut-name=AonoKaname en-aut-sei=Aono en-aut-mei=Kaname kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=14 ORCID= en-aut-name=NagashimaHideo en-aut-sei=Nagashima en-aut-mei=Hideo kn-aut-name= kn-aut-sei= kn-aut-mei= aut-affil-num=15 ORCID= affil-num=1 en-affil= kn-affil=Okayama University affil-num=2 en-affil= kn-affil=Okayama University affil-num=3 en-affil= kn-affil=Okayama University affil-num=4 en-affil= kn-affil=Okayama University affil-num=5 en-affil= kn-affil=Okayama University affil-num=6 en-affil= kn-affil=Okayama University affil-num=7 en-affil= kn-affil=Okayama University affil-num=8 en-affil= kn-affil=Okayama University affil-num=9 en-affil= kn-affil=Okayama University affil-num=10 en-affil= kn-affil=Okayama University affil-num=11 en-affil= kn-affil=Okayama University affil-num=12 en-affil= kn-affil=Okayama University affil-num=13 en-affil= kn-affil=Okayama University affil-num=14 en-affil= kn-affil=Okayama University affil-num=15 en-affil= kn-affil=Okayama University en-keyword=computed tomographic arteriography kn-keyword=computed tomographic arteriography en-keyword=hepatocellular carcinoma kn-keyword=hepatocellular carcinoma en-keyword=hepatic arteriography kn-keyword=hepatic arteriography en-keyword=contrast enhancement kn-keyword=contrast enhancement END